Review – Kaplan Pediatrics: Disorders of the Eye

ABNORMALITIES OF THE EYE STRUCTURES

Pupils and iris

Coloboma of iris

Often autosomal dominant

Defect of lid, iris, lens, retina, or choroid

Always inferior—keyhole appearance of iris; in lid, manifests as cleft

Possible CHARGE association

Leucokoria—white reflex

Retinoblastoma

Cataract

Retinopathy of prematurity

Retinal detachment

Larval granulomatosis

 

 

Lens

 

 

Cataracts—lens opacities; the most important congenital etiologies:

Prematurity (many disappear in a few weeks)

Inherited—most autosomal dominant

Congenital infection—TORCH (especially rubella); also, measles, polio, influenza, varicella, vaccinia

Galactosemia

Chromosomal (trisomies, deletions and duplications, XO)

Drugs, toxins, and trauma (steroids, contusions, penetrations)

Ectopia lentis—instability or displacement of lens; edge of displaced lens may be visible in pupillary aperture

Differential:

Trauma—most common

Uveitis, congenital glaucoma, cataract, aniridia, tumor

Systemic causes

} Marfan syndrome (most with superior and temporal; bilateral)

} Homocystinuria—inferior and nasal

} Ehlers-Danlos

Ocular muscles

Strabismus

Definition—Misalignment of the eyes from abnormal innervation of muscles

Diagnosis—Hirschberg corneal light reflex—most rapid and easily performed;

light reflex should be symmetric and slightly nasal to center of each pupil

Patch the good eye to eliminate amblyopia, the eye muscle surgery

Pseudostrabismus

Epicanthal folds and broad nasal bridge

Caused by unique facial characteristics of infant

Transient pseudostrabismus; common up to age 4 months

 

Conjunctiva

A 12-hour-old newborn is noted to have bilateral conjunctival injection, tearing, and some swelling of the left eyelid. Physical examination is otherwise normal.

 

Ophthalmia neonatorum

Redness, chemosis, edema of eyelids, purulent discharge

Causes:

Chemical conjunctivitis most common in first 24 hours of life

From silver nitrate and erythromycin

  1. gonorrhea2–5-day incubation; may be delayed >5 days due to suppression from prophylactic eye treatment

} Mild inflammatory and serosanguineous discharge, then thick and purulent

} Complications—corneal ulceration, perforation, iridocyclitis

  1. trachomatis5–14-day incubation; most common

} Mild inflammation to severe swelling with purulent discharge

} Mainly tarsal conjunctivae; cornea rarely affected

Diagnosis—Gram stain, culture, PCR (polymerase chain reaction) for chlamydia

Treatment:

  1. gonorrhea: ceftriaxone ´ 1 dose IM + saline irrigation until clear
Chlamydia: erythromycin PO ´ 2 weeks + saline irrigation until clear (may prevent subsequent pneumonia)

The red eye

Bacterial conjunctivitis

General conjunctival hyperemia, edema, mucopurulent exudate (crusting of lids together), and eye discomfort

Unilateral or bilateral

  1. pneumonia, H. influenza (non-typable), S. aureus, other strep

Treatment—warm compresses and topical antibiotics

Viral conjunctivitis

Watery discharge, bilateral, usually with URI

Adenovirus, enterovirus

Epidemic keratoconjunctivitis = adenovirus type 8

Good hand-washing

Allergic

Chemical

Household cleaning substances, sprays, smoke, smog

Extensive tissue damage, loss of sight

Keratitis—corneal involvement

  1. simplex, adenovirus, S. pneumoniae, S. aureus, pseudomonas, chemicals

Foreign bodies → corneal abrasion (pain, photophobia)

Anterior uveitis = iridocyclitis (from ciliary body to iris)

Periorbital versus orbital cellulitis (see below)

Dacryocystitis (S. aureus, H. influenza, S. pneumoniae), dacroadenitis (S. aureus, streptococci, CMV [cytomegalovirus], measles, EBV [Epstein-Barr virus], trauma)

Treatment—underlying cause and topical steroids

 

Retina and vitreous

Retinopathy of prematurity (ROP)

Prematurity, hyperoxia, and general illness

From mild to severe progressive vasoproliferative scarring and blinding retinal detachment

Treatment—cryosurgery or laser photocoagulation

Retinoblastoma

Most common primary malignant intraocular tumor

Recessive-suppressive gene—13q14 → family members need to be screened

Average age of diagnosis = 15 months for bilateral and 25 months for unilateral

Rarely discovered at birth

Initial sign in most = leucokoria

Appears as white mass

Second most common—strabismus

Diagnosis—CT scan to confirm; no biopsy (spreads easily)

Need to consider enucleation—radiation, chemotherapy, laser therapy, cryotherapy

Prognosis poor if extends into orbit or optic nerve

 

 

EYE INJURIES

Corneal abrasions

Symptoms—pain, tearing, photophobia, decreased vision

Diagnosis—first anesthetize eye, then fluorescein and blue-filtered light (Wood’s lamp)

Treatment—pain relief and topical antibiotics

 

Foreign body

Topical anesthetic and irrigation to remove

If embedded, send to ophthalmologist

 

PERIORBITAL VERSUS ORBITAL CELLULITIS

Periorbital cellulitis

Inflammation of lids and periorbital tissue without signs of true orbital involvement; insidious onset; low-grade fever; no toxicity

Causes—trauma, infected wound, abscess of lid, sinusitis, bacteremia (H. influenza

nontypable, S. pneumoniae, S. aureus)

May be first sign of sinusitis that may progress to orbital cellulitis

Physical exam: inflammation with intact eye movements; normal vision; no proptosis

Diagnosis—clinical (blood culture unlikely to be positive)

Treatment—oral or IV (depending on severity) antibiotics (cover for S. aureus and gram positive resistant strains)

Orbital cellulitis

A 7-year-old boy presents with swelling around the eye 2 days after suffering an insect bite to the eyelid. There is edema, erythema, and proptosis of the eye. Marked limitation of eye movements are noted. He has a low-grade fever.

 

Infection of orbital tissue including subperiosteal and retrobulbar abscesses

Physical examination

Ophthalmoplegia (eyeball does not move)

Chemosis

Inflammation

Proptosis

Toxicity, fever, leukocytosis, acute onset

Causes: paranasal sinusitis, direct infection from wound, bacteremia

Organisms nontypable H. influenza, S. aureus, beta hemolytic strep, S. pneumoniae, anaerobes

Diagnosis—CT scan with contrast of orbits and surrounding area (best initial test)

Treatment—Intravenous antibiotics (again, cover for S. aureus) and may require sinus and/or orbital drainage (will give you culture and sensitivities) if no improvement