Review – Kaplan Pediatrics: Orthopedic Disorders

Review – Kaplan Pediatrics: Orthopedic Disorders

DISORDERS OF THE HIP

 

Developmental Dysplasia of the Hip (DDH)

  • General ligamental laxity

Family history

  • Significantly more females
  • Firstborn
  • Breech
  • Oligohydramnios
  • Multiple gestation
  • Physical examination
    • Barlow is most important examination; will dislocate an unstable hip; is easily felt (clunk not a click)
    • Ortolani—reduces a recently dislocated hip (most at 1–2 months of age), but after 2 months, usually not possible because of soft-tissue contractions
  • Diagnosis–ALL (+) → refer immediately for orthopedic consult If unsure:
−   Dynamic ultrasound of hips best test age <4 months
  • After 4 months, frog lateral x-ray
  • Treatment
    • Pavilk harness for 1–2 months
    • Surgery, casting
  • Complicationsacetabular dysplasia, leg length discrepancy

 

Legg-Calvé-Perthes Disease

A 5-year-old boy has developed progressive limping. At first painless, it now hurts to run and walk. The pain is in the anterior thigh. The pain is relieved by rest. Parents recall no trauma.

 

  • Idiopathic avascular necrosis of the capital femoral epiphysis in immature, growing child
  • More in males; 20% bilateral; sometimes after trauma
  • Presentation—mild intermittent pain in anterior thigh with painless limp with restriction of motion

 

 

  • Diagnosis—anterior/posterior and frog leg lateral x-ray shows compression, collapse, and deformity of femoral head
  • Treatment
    • Containment (femoral head within acetabulum) with orthoses or casting
    • Bedrest
    • Abduction stretching exercises
    • If significant femoral deformity persists, surgical correction

©2007 Kaplan Medical. Reproduced with permission from Dr. Philip Silberberg, University of California at San Diego

 

Figure 17-1. MRI Demonstrating Legg-Calve-Perthes Disease

 

 

Slipped Capital Femoral Epiphysis (SCFE)

  • Most common adolescent hip disorder
  • Either obese with delayed skeletal maturation, or thin with a recent growth spurt
  • Can occur with an underlying endocrine disorder
  • Clinical presentation
    • Pre-slip stable; exam normal; mild limp external rotation
    • Unstable slip; sudden-onset extreme pain; cannot stand or walk; 20% complain of knee pain with decreased hip rotation on examination
  • Complications—osteonecrosis (avascular necrosis) and chondrolysis (degeneration of cartilage)
  • Diagnosis—AP and frog-leg lateral x-ray, earliest finding: widening of physis with- out slippage (preslip); as slippage occurs, femoral neck rotates anteriorly while head remains in acetabulum
  • Treatment—open or closed reduction (pinning)

 

 

 

©2007 Kaplan Medical. Reproduced with permission from Dr. Philip Silberberg, University of California at San Diego

 

Figure 17-2. X-ray of the Hips Demonstrating Slipped Capitol Femoral Epiphysis

 

 

Transient Synovitis

  • Viral; most 7–14 days after a nonspecific upper respiratory infection; most at 3–8 years of age
  • Clinical presentation
    • Acute mild pain with limp and mild restriction of movement
    • Pain in groin, anterior thigh, and knee
  • Diagnosis
    • Small effusion (±)
    • Slight increase in ESR
−   Normal x-rays
  • No to low-grade fever; non-toxic-appearing
  • Treatment—bedrest and no weight-bearing until resolved (usually <1 week), then 1–2 weeks of limited activities

 

 

INTOEING

 

Metatarsus Adductus

  • Most common in firstborn (deformation)
  • Forefoot adducted from flexible to rigid
  • Treatment—primarily nonsurgical; serial plaster casts before 8 months of age; ortho- ses, corrective shoes; if still significant in a child age >4 years, may need surgery

 

 

 

Note

In talipes equinovarus, the patient’s heel can’t go flat on the exam surface (as opposed to metatarsus adductus, in which the heel can).

Talipes Equinovarus (Clubfoot)

A newborn is noted to have a foot that is stiff and slightly smaller than the other one. The affected foot is medially rotated and very stiff, with medial rotation of the heel.

 

  • Congenital, positional deformation, or associated with neuromuscular disease
  • Hindfoot equinus, hindfoot and midfoot varus, forefoot adduction (at talonavicular joint)
  • Treatment
    • Complete correction should be achieved by 3 months (serial casting, splints, ortho- ses, corrective shoes); if not, then surgery

 

Internal Femoral Torsion (Femoral Anteversion)

  • Most common cause of intoeing ³2 years of age; entire leg rotated inwardly at hip during gait
  • Most are secondary to abnormal sitting habits (W-sitting).
  • Treatment—observation; takes 1–3 years to resolve; surgery only if significant at >10 years of age

 

Internal Tibial Torsion

  • Most common cause of intoeing <2 years of age (also because of in utero position- ing); often with metatarsus adductus
  • Measure prone thigh/foot angles
  • No treatment needed—resolves with normal growth and development; takes 6–12 months (is physiologic)

 

 

DISORDERS OF THE KNEE

 

Osgood-Schlatter Disease

  • Traction apophysitis of tibial tubercle (overuse injury)
  • Look for active adolescent (running, jumping)
  • Swelling, tenderness, increased prominence of tubercle
  • Treatment—rest, restriction of activities, knee immobilization, isometric exercises
  • Complete resolution requires 12–24 months

 

 

DISORDERS OF THE SPINE

 

Scoliosis

A 12-year-old girl is seen for routine physical examination. She voices no complaints. Examination is remarkable for asymmetry of the posterior chest wall on bending forward. One shoulder appears higher than the other when she stands up.

 

 

 

  • Most are idiopathic; rarely, hemivertebra
  • Others are congenital, with neuromuscular disorders, compensatory, or with intraspi- nal
  • Slightly more females than males; more likely to progress in females
  • Adolescent (>11 years) more common
  • Adams test bending forward at hips —almost all with >20-degree curvature are identified in school screening programs (but many false positives)
  • Diagnosis—x-ray is standard: posterior/anterior and lateral of entire spine gives great- est angle of curvature
  • Treatment—trial brace for immature patients with curves <30–45 degrees and surgery for those >45 degrees (permanent internal fixation rods)

 

DISORDERS OF THE UPPER LIMB

Nursemaid Elbow

  • When longitudinal traction causes radial head subluxation
•        History of sudden traction or pulling on arm
  • Physical exam reveals a child who refuses to bend his/her arm at the elbow
  • Treatment—rotate hand and forearm to the supinated position with pressure of the radial head → reduction

 

OSTEOMYELITIS AND SEPTIC ARTHRITIS

  • Etiology
−   Osteomyelitis:
  • aureus most common overall, in all
  • Pseudomonas—puncture wound
  • More Salmonella in sickle cell ( aureus still most common)
−   Septic arthritis:
  • Almost all aureus
  • Most in young children; hematogenous; LE > UE and other parts of body
  • Presentation
    • Pain with movement in infants
    • Older—fever, pain, edema, erythema, warmth, limp, or refusal to walk (acute, toxic, high fever)

Note

  • rays for patients with osteomyelitis are initially Changes are not seen until 10−14 days.

 

 

  • Diagnosis
    • Blood culture, CBC, ESR
  • Radiographic studies:
    • Initial plain film if diagnosis not obvious to exclude other causes—trauma, foreign body, tumor; trabecular long bones do not show changes for 7−14 days (septic arthritis shows widening of joint capsule and soft-tissue edema)
    • Ultrasound for septic arthritis—joint effusion, guide localization of drainage
    • Best test is MRI for osteo; very sensitive and specific
    • Bone scan—can be valuable to augment MRI, especially if multiple foci are suspected or vertebrate
  • Definitive—aspirate for culture and sensitivity
    • Osteomyelitis → bone biopsy for culture and sensitivity
    • Septic arthritis → ultrasound guided arthrocentesis for culture and sensitivity
    • Treatment
      • Intravenous antibiotics—always cover for Staphylococcus initially (treatment for osteo much longer)

 

 

OSTEOGENESIS IMPERFECTA

  • Susceptibility to fracture of long bones or vertebral compression from mild trauma
  • Most common genetic cause of osteoporosis; all types caused by structural or quan- titative defects in type I collagen
•        Autosomal dominant
  • Clinical triad is fragile bones, blue sclera, and early deafness (and short stature)
  • Four types, from perinatally lethal
    • to very severe, nonlethal
  • Diagnosis
    • May see fractures on prenatal ultrasound as early as 6 weeks

– Rule out child abuse due to fracture and injury history.

  • Confirmed by collagen biochemical studies using fibroblasts cultured from a skin- punch biopsy
  • Treatment—no cure; physical rehabilitation; fracture management and correction of deformities

Courtesy of Tom D. Thacher, M.D.

 

Figure 17-3. Blue Sclera in Osteogenesis Imperfecta

 

 

 

 

Courtesy of Tom D. Thacher, M.D.

 

Figure 17-4. Skeletal Malformation Due to Osteogenesis Imperfecta

 

 

BONE TUMORS

 

Table 17.1. Comparison of Osteogenic Sarcoma, Ewing Sarcoma, and Osteoid Ostcoma
Osteogenic Sarcoma Ewing Sarcoma Osteoid Osteoma
Presentation Second decade Second decade Second decade
 

M:F

Slightly greater in males Slightly greater in males  

3x greater in males

 

Predisposition

Retinoblastoma, radiation  

None

 

Male gender

 

X-ray

 

Sclerotic destruction:

“sunburst”

Lytic with laminar periosteal eleva- tion: “onion skin” Small round central lucency with sclerotic margin
Malignant Yes Yes No
Metastases Lungs, bone Lungs, bone N/A
 

Treatment

 

Chemotherapy, ablative surgery

 

Radiation and/or surgery

NSAIDs

Surgery recommended when associated pain

 

Prognosis

70% cure without metastasis at diagnosis 60% cure without metastasis at diagnosis  

Over time it may resolve spontaneously

Outcome if metastasis ≤20%  

20–30%

 

N/A